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Once in the body, semaglutide binds to GLP-1 receptors in the hypothalamus, the region of the brain responsible for regulating hunger and satiety
This mechanism is why slow-release carbohydrates and prebiotic fibers are increasingly positioned to raise endogenous GLP-1 and PYY as a non-drug route to appetite regulation
The collaborative action of these compounds presents a holistic strategy that holds promise for potentially addressing conditions associated with inflammation, including arthritis, inflammatory bowel disease, and tendonitis
