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Exocrine pancreatic insufficiency (EPI) prevents animals from being able digest and absorb the nutrients from food, due to lack of digestive enzymes in the pancreas
After subcutaneous administration, it circulates systemically, reaches multiple injury locations simultaneously, and modulates the inflammatory environment throughout the body rather than at a single point
It incorporates key modifications, including 14E/17R mutations to stabilize an -helical segment via a salt bridge, multiple proline substitutions (25P, 28P, 29P) to reduce -sheet formation, and N-terminal C20 fatty diacid lipidation for reversible albumin binding (Kruse et al
doi: 10.1016/j.tins.2013.01.005 46 KarlssonFHUsseryDWNielsenJNookaewI
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